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. 2020 Apr 15;20:122. doi: 10.1186/s12935-020-01188-w

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Tenascin-C (TNC) knockdown exhibited dramatically decreased LSD1 expression. a, b Protein expression of TNC in the HT29 and HCT116 cells after transfecting with nuclease-free water (blank control), control siRNA and TNC siRNA (s7068, s7069 and s7070) for 48 h was confirmed by western blotting analysis. c, d Protein expression of LSD1 in the HT29 and HCT116 cells after transfecting with TNC siRNA compared with control siRNA. Western blotting data were normalized to those for β-actin and expressed as fold changes relative to levels in the control group. **P < 0.01, ***P < 0.001 versus control