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. 2010 Mar 9;110(1):118–128. doi: 10.1002/jcb.22518

Table II.

Respective Reference Genes and Cellular Genes Encoding RV Interaction Partners and Viral Genes With Respective Data for RT‐qPCR

Gene Amplicon size (bp) ESlope (%)a ELinReg (%)b Ct Ct of RT −vec P (bp)c
Candidate reference genes
 TBP 226 86 84 25
 HPRT1 94 90 88 20
 PPIA 325 96 73 20 >36 +(602)
 HUEL 104 94 86 24
 B2MG 92 89 73 20 >36
 β‐actin 317 82 78 18 >36 +(506, 296)
 GAPDH 86 95 89 19 31 +(67)
 RPII 632 95 82 28 +(609)
 ECHS 200 89 85 22 >36
Genes possibly relevant for RV replication
 p32 77 n.d. 90 19 >36
 p53 77 n.d. 89 25 33
 RB 76 n.d. 87 24 >36
 p21 67 n.d. 87 27 >36
 PABP 96 n.d. 70 22 28
 SLC25A4 100 n.d. 87 21 +(994)
 SP100 110 n.d. 82 19 +(1129)
Target on RV genome
 P150 90 n.d. 84 variable >36

n.d., not determined; Ct, cycle threshold; RT −ve, minus RT control.

a

10‐fold serial dilutions of cDNA obtained from Vero cells were plotted against dilution factors. RT‐qPCR efficiencies (E) were calculated by the following equation (Rasmussen, 2001): E = 10(−1/slope). Only Ct values < 40 were included.

b

PCR efficiency was calculated based on the starting point of the exponential phase of amplification using LinReg PCR program.

c

The Ct values of RT −ve samples appear to be due to retropseudogenes (P) that lead to amplification of contaminating genomic DNA and could thus possibly interfere with RT‐qPCR results. If present, melting peaks for the RT−ve samples were distinguishable from the specific amplicon. The possible amplification of retropseudogenes with the primer sequences used in this study was determined by BLAT search [Kent, 2002. −, no retropseudogenes; +, retropseudogenes present, yielding amplicons of the indicated length.