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. 2019 Nov 6;235(4):3280–3291. doi: 10.1002/jcp.29104

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© 2019 Wiley Periodicals, Inc.

This article is being made freely available through PubMed Central as part of the COVID-19 public health emergency response. It can be used for unrestricted research re-use and analysis in any form or by any means with acknowledgement of the original source, for the duration of the public health emergency.

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Inhibition of FHL3 expression could reverse the antitumor actions induced by the knockdown of poly (C)‐binding protein 2 (PCBP2) in glioma cells in vitro. Compared to the control groups, the cells proliferation (a), migration (b) and invasion (c) were significantly enhanced, while cell apoptosis (d) was downregulated in + si‐FHL3 group. The cells in control group were transfected by si‐PCBP2 vector only, while the cells in + si‐FHL3 group were cotransfected by si‐PCBP2 and si‐FHL3 vectors. Data are presented as mean ± SD (n = 3). *p < .05, **p < .01, and ***p < .001. SD, standard deviation