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. 2005 May 13;206(3):251–259. doi: 10.1002/path.1769

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Copyright © 2005 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.

This article is being made freely available through PubMed Central as part of the COVID-19 public health emergency response. It can be used for unrestricted research re-use and analysis in any form or by any means with acknowledgement of the original source, for the duration of the public health emergency.

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Lung alveoli of a normal healthy rhesus monkey for comparison. (A) Normal alveolar septa (H&E; original magnification ×200). (A, a) The inter‐alveolar septa are narrow; there is no inflammatory cell infiltration or oedema (H&E; original magnification ×200). (A, b) The alveolar sacs are clear; no inflammatory cells are seen in any of the alveoli shown (H&E; original magnification ×200). Panel B, a shows intact reticulin fibres in the alveolar septa; B, b shows small capillaries located in the inter‐alveolar septa (MCT; original magnification ×400). Panel C shows the intact lining epithelium; C, a, b shows positive staining of epithelial cells with a monoclonal anti‐cytokeratin antibody (CK +; original magnification ×1000). Panel D, a, b shows that foam cells staining positive for CD68 are adherent to regenerating epithelial cells (MAb against human CD68; original magnification ×1000)