Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2017 Mar 30;85(4):280–290. doi: 10.1111/sji.12532

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2017 The Foundation for the Scandinavian Journal of Immunology

This article is being made freely available through PubMed Central as part of the COVID-19 public health emergency response. It can be used for unrestricted research re-use and analysis in any form or by any means with acknowledgement of the original source, for the duration of the public health emergency.

PMC Copyright notice

Effect of different acute respiratory infections in the cytokine/chemokine production. Viral and mixed (viral and bacterial) infections induced higher production of interleukin‐1 beta (IL‐1β), tumour necrosis factor‐alpha (TNF‐α), monocyte chemoattractant protein‐1 (MCP‐1) and regulated on activation normal T cell expressed and secreted factor (RANTES) than those observed in bacterial infection (A, B, E, F). High values of interleukins 4 and 5 (IL‐4 and IL‐5) were observed, but they remained similar in all types of infections (C, D). Viral (n = 21); bacterial (n = 11); mixed (n = 11); control (n = 10). These data were obtained from asthmatic and non‐asthmatic patients. Data were analysed using a nonparametric ANOVA (Kruskal–Wallis test) and Dunn's multiple comparisons test. Values were expressed as mean ± standard deviation.