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. 2020 Mar 31;8(1):e001120. doi: 10.1136/bmjdrc-2019-001120

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© Author(s) (or their employer(s)) 2020. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/.

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Pathological changes in the innermost retina in the diabetic model mouse. At the age of 20 weeks, there was a significant loss of ganglion axon bundles labeled with Tubulin β3 (A, B) and a significant decrease in Laminin thickness in the innermost retina (C, D) in diabetic βPdk1^–/– mice compared with control Pdk1^flox/flox mice. DM, diabetes mellitus.