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. 2020 Apr 20;11(4):256. doi: 10.1038/s41419-020-2424-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a–c IHC of liver sections (magnification ×400) from mice with I/R injury treated with UC-MSCs or PBS and killed at 6 and 24 h after reperfusion were stained with TOM20 (column 1), Parkin (column 2) and PINK1 (column 3) antibody. The IHC staining of PINK1, Parkin and TOM20 in liver tissues was semiquantified (n = 5/group). d The expression of Parkin, PINK1 and TOM20 in liver tissues was determined by western blotting, and the average intensities of the bands in the western blot were quantified (n = 5/group). The data are presented as the means ± SEMs. *p < 0.05, **p < 0.01, ***p < 0.001 (all p-values were obtained by one-way ANOVA).