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. 2020 Apr 21;20:120. doi: 10.1186/s12906-020-02902-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Effect of astragalin on Nrf2 signaling pathway in rat brain with I/R injury. a Nrf2 (nucleus) and HO-1 protein expression levels. b Quantitative results of Nrf2/Lamin B1 protein expression. c Quantitative results of HO-1 protein expression. d SOD activity of each group of rats. e Content of GSH in each group of rats. f Content of GSH-Px in each group of rats. Data represent the average ± SD of three independent experiments for each bar. n = 10.Compared with the sham operation group, ##P < 0.01; compared with the I/R group, *P < 0.05, **P < 0.01