Skip to main content
. 2017 Nov 27:139–178. doi: 10.1016/B978-0-12-801238-3.10923-7

Table 2.

Defense mechanisms of the gastrointestinal system

Taste buds
Vomiting
Saliva
Flushing action, so potential pathogens are cleared from the oropharynx
Protective coating of the mucosa
Contains antimicrobial lysozyme, lactoferrin, lactoperoxidase, and immunoglobulins
Gastric pH
Microbiota/microbiome—lower GI (damaged by toxicants; carcinogen activation)
100 trillion (anaerobic) bacteria (10 × host); 3.3 million genes (150 × host)
Bacteriocins
Compete for nutrients
Compete for attachment sites
Promote immune system maturation
Biotransformation
Enterotype
Secreted immunoglobulins
Extraintestinal secretions from the liver and pancreas
Lactoferrins
Peroxidase
Intestinal proteolytic enzymes
Intestinal biotransforming and metabolic enzymes
Phagocytes and other effector cells within the submucosa
High rate of epithelial turnover
Shedding of receptor laden ALP and catalase containing vesicles from microvilli
Large surface area
Dilution with ingesta
Increased peristalsis resulting in diarrhea
Mucus—contains phages that destroy bacteria > 1 × 104
Paneth cells (antimicrobial peptides, lysozymes, phospholipase A2, defensins–cryptdins)
Innate lymphoid cells
Adaptive immune system
Kupffer cells (liver)
Genetic polymorphisms (HLA) and host gene expression

Reproduced from Zachary, J. F. and McGavin, M. D. (2016). Pathologic basis of veterinary disease (6th edn.). Copyright © 2016 by Mosby, Inc, an affiliate of Elsevier Inc.