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. 2020 Apr 22;5:44. doi: 10.1038/s41392-020-0137-7

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — NUSAP1 is highly expressed in GBM patients and in GBM cells. a Immunohistochemical analyses of NUSAP1 in normal brains and glioma tissues. b Statistical analyses of NUSAP1 in 8 normal brains and 64 glioma tissues. c The expression of NUSAP1 in normal brain as well as in glioma patients and in cell lines from 13 different data sets. d Analyses of NUSAP1 in living patients and in dead patients from three different databases. e The level of NUSAP1 in five GBM cell lines as well as in a pair of peritumoral and tumor tissues. f Kaplan–Meier analysis of progression-free survival using data from three different glioma databases