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. 2020 Apr 15;12:86. doi: 10.3389/fnagi.2020.00086

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Copyright © 2020 Wang, Yang, Wang, Li, Li, Sun, Zhao, Ma, Hua and Yang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Astrocyte proliferation and neuronal apoptosis in the hippocampal CA1 region of BCAS and BCAO rats (A). GFAP and NeuN expression in the hippocampal CA1 area 30 days after surgery (B and C). The endpoint, the process length and average area of astrocytes. (D–G) Data are expressed as the mean ± SD (n = 6/group). ^∗P < 0.05 compared with the control group; ^#P < 0.05 compared with the BCAS group. BCAS, bilateral common carotid artery stenosis; BCAO, bilateral common carotid artery occlusion; SD, standard deviation; GFAP, glial fibrillary acidic protein; DAPI, 4’,6-diamidino-2-phenylindole.