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. 2020 Feb 28;58(1):4–8. doi: 10.5114/reum.2020.93505

Table II.

Clinical manifestations depending on the incidence of the type of anti-ARS antibodies (ASA) in the group of 50 analyzed patients

Type of antibody n Muscle weakness n (%) Body mass loss n (%) Joint involvement n (%) Pulmonary involvement n (%) Skin involvement n (%) Heart involvement n (%) Dysphagia n (%) CK elevation n (%) CRP elevation n (%) Fever n (%)
Jo-1 23 11 (47.8) 2 (8.7) 15 (65.2) 11 (47.8) 14 (60.8) 2 (8.7) 1 (4.3) 10 (43.4) 10 (43.4) 6 (26)
PL-7 8 5 (62.5) 3 (37.5) 4 (50) 5 (62.5) 3 (37.5) 1 (12.5) 2 (25) 3 (37.5) 2 (25) 3 (37.5)
PL-12 16 10 (62.5) 4 (25) 13 (81.25) 8 (50) 5 (31.25) 1 (6.25) 4 (25) 5 (31.25) 6 (37.5) 4 (25)
OJ 6 4 (66.6) 2 (33.3) 3 (50) 6 (100) 4 (66.6) 1 (16.6) 2 (33.3) 4 (66.6) 3 (50) 3 (50)
EJ 3 1 (33.3) 0 3 (100) 1 (33.3) 2 (66.6) 0 0 0 1 (33.3) 1 (33.3)

In 6 patients the coexistence of at least one ASA was found. Anti-ARS – anti-aminoacyl tRNA synthetase, ASA – antisynthetase antibodies, CK – creatine kinase, CRP – C-reactive protein, Jo-1 – anti-histidyl tRNA synthetase, PL-7 – anti-threonyl tRNA synthetase, PL-12 – anti-alanyl tRNA synthetase, OJ – anti-isoleucyl tRNA synthetase, EJ – anti-glycyl tRNA synthetase.