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. 2020 Apr 11;7:501–509. doi: 10.1016/j.toxrep.2020.04.001

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© 2020 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 3 — The effects of benomyl on redox status of SH-SY5Y cells were evaluated by measuring the levels of reactive oxygen species (ROS) using H2DCF-DA (a and b) and GSH with the DTNB assay (c). Median florescence intensities [MFIs] were shifted to right (a, representative histogram) indicating that ROS production was significantly increased at all tested concentrations compared to the control (b, bar graphs). GSH concentration that is expressed as μg/mg protein remained unaffected (c). The control cells were exposed to 1% DMSO. Error bars represent the standard deviation. *: p < 0.05; †: p < 0.01 compared to the control group.

Fig. 3 — The effects of benomyl on redox status of SH-SY5Y cells were evaluated by measuring the levels of reactive oxygen species (ROS) using H2DCF-DA (a and b) and GSH with the DTNB assay (c). Median florescence intensities [MFIs] were shifted to right (a, representative histogram) indicating that ROS production was significantly increased at all tested concentrations compared to the control (b, bar graphs). GSH concentration that is expressed as μg/mg protein remained unaffected (c). The control cells were exposed to 1% DMSO. Error bars represent the standard deviation. *: p < 0.05; †: p < 0.01 compared to the control group.