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. 2020 Apr 21;20:187. doi: 10.1186/s12872-020-01474-5

Table 2.

Laboratory data and incidence of CI-AKI (n (%) or median [IQR]) (n = 284)

metformin
n = 119
No Metformin
n = 165
p value
HbA1c(%) 7.0 [3.6–14.2] 7.8 [6.9–8.8] 0.194
Hb(g/l) 141 [126–152] 120 [110–140] 0.079
cTnI peak (ng/ml) 7.0 [3.6–14.2] 7.8 [6.9–8.8] 0.842
Creatinine
 Baseline (μmol/l) 76 [66–86] 73 [61–84] 0.105
 Post PCI (μmol/l) 83 [72–98] 76 [64–76] 0.012
 Relative creatinine change 11% [6–20%] 11% [5–22%] 0.858
post PCI (%)
  ≥ 50% increase 5 (4.2%) 9 (5.5%) 0.630
 Absolute creatinine change 7.9 [2.4–13.2] 6.7 [0.2–12.7] 0.198
post PCI (%) (μmol/l)
  ≥ 27 μmol/l(0.3 mg/dl) 15 (12.6%) 15 (9.1%) 0.342
eGFR (ml/min/1.73m2)
 Baseline (ml/min/1.73m2) 89 [73–104] 94 [72–113] 0.170
 eGFR≤60 ml/min/1.73m2 9 (7.6%) 21 (12.7%) 0.163
 Post PCI (ml/min/1.73m2) 77 [65–95] 87 [67–102] 0.092
 Relative eGFR change 13.4% [6.4–25.1%] 13.5% [5.2–24.3%] 0.449
post PCI (%)
 Absolute eGFR change −7 [−16; 3] −6 [−18; 3] 0.981
post PCI (ml/min/1.73m2)
 CI-AKI 15 (12.6%) 17 (10.3%) 0.545

HbA1c glycosylated hemoglobin, Hb hemoglobin, eGFR glycosylated hemoglobin estimated glomerular filtration rate, cTnI cardiac troponin I, PCI percutaneous coronary intervention, CI-AKI contrast induced-acute renal injury