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. 2020 Apr 21;18:177. doi: 10.1186/s12967-020-02337-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Previous MM therapy. Previous anti-MM therapy (middle row), their respective time intervals (left row) and disease responses (right row) before onset of PML. The patient received seven lines of therapy, including a tandem high dose (HD) melphalan therapy and an allo-HCT with successive interferon and donor lymphocyte infusions (DLI). The last line of therapy before JC virus reactivation contained Pomalidomide (Pom), Bortezomib, Cyclophosphamid and Daratumumab (Dara)