Figure 6.

IL‐17 stimulates ECs dysfunction upon lipopolysaccharide (LPS) exposure. A‐D, IL‐17 inhibited cell viability (A) (*P < .05, n = 5), improved lactate dehydrogenase (LDH) release (B) (*P < .05, n = 5) and increased apoptosis in ECs subjected to inflammation induced by LPS (C, D) (*P < .05, n = 3). E and G, Western blot analysis of the expression of cleaved caspase‐3 and Bim (*P < .05, n = 3). F, The production of chemokine and adhesive molecules were determined by ELISA (*P < .05, n = 3)