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(A) Staged approach of potential mitochondrial targets to attenuate Dox-induced cardiotoxicity, ranging from acute to more chronic stages after Dox administration. (B) Underlying mitochondrial and cell death mechanisms leading to Dox-induced cardiotoxicity. Bnip3, Bcl-2/19 kDa interacting protein 3; mPTP, mitochondrial permeability transition pore; NF-κB, nuclear factor-κB.
