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. Author manuscript; available in PMC: 2021 May 1.
Published in final edited form as: Arterioscler Thromb Vasc Biol. 2020 Mar 12;40(5):1256–1274. doi: 10.1161/ATVBAHA.120.314147

Figure 8. Differential expression of LMCD1 in human and mouse neointimal hyperplasia.

Figure 8.

A. Human stenotic and non-stenotic artery sections were stained for SMCα-actin (green) and LMCD1 (red). The sections were also stained with normal mouse or rabbit IgG as negative controls. B. Uninjured and 3 weeks of post guidewire-injured mouse femoral artery cross-sections were stained for SMCα-actin (red) and LMCD1 (green). C. Tissue extracts prepared from uninjured and guide wire-injured mouse femoral arteries were analyzed by Western blotting for LMCD1, CDC6 and IL-33 levels using their specific antibodies and normalized to α-tubulin levels. The bar graphs represent Mean ± S.D. values of six animals (two pooled arteries for each Western blotting analysis). *, p < 0.05 versus uninjured. Scale bar is 50 μm.