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. 2020 Apr 24;11(4):273. doi: 10.1038/s41419-020-2478-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Protein levels of DDX11 in 8 paired HCC and matched adjacent non-tumor tissues were determined by western blot assay. (N, matched adjacent non-tumor tissues; T, tumor tissues). b Representative expression pattern of DDX11 in HCC tissue samples. c Distribution of DDX11 immunohistochemical staining scores in HCC tissue samples according to TNM stage, histological grade, tumor size or vascular invasion. d, e Representative expression pattern of DDX11 in HCC tissue samples from patients with different TNM stages or in HCC tissue samples from patient with absent/present vascular invasion. f Univariate analysis of the association between DDX11 expression and clinicopathological features. g Kaplan–Meier analysis of the overall survival (OS) rate in HCC patients with high- or low- DDX11 expression in ZZU HCC cohort. *p < 0.05, **p < 0.01, ***p < 0.001.