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. 2020 Apr 24;3:193. doi: 10.1038/s42003-020-0933-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a, b MTT-based cytotoxicity of KH-3 against a panel of TNBC cell lines (a) and MDA-MB-231 cells, sgControl and two HuR KO clones (b). c–h Half-life of Bcl-2, Msi2 and XIAP mRNA in MDA-MB-231 (c–e) and SUM159 (f–h) cells treated with 5 μg/mL actinomycin D together with DMSO, KH-3 or KH-3B. Data are mean ± SD from n = 3 independent experiments (*P < 0.05, **P < 0.01, ***P < 0.001, two-way ANOVA).