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. 2020 Apr 21;26(15):1758–1774. doi: 10.3748/wjg.v26.i15.1758

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©The Author(s) 2020. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Mdivi-1 administration inhibits mitochondrial fission and reactive oxygen species generation in Caco-2 hypoxia/reoxygenation cells. A: Deep Red FM in Caco-2 cells (scale bar = 10 μm); B, C: Dynamin-related protein 1 and dynamin-related protein 1 Ser637 expression upon mdivi-1 administration (n = 3); D: MitoSOX assay in Caco-2 cells (scale bar = 10 μm). ^cP < 0.001 compared with the control group; ^eP < 0.01 compared with the hypoxia/reoxygenation group. DRP1: Dynamin-related protein 1; PINK1: PTEN induced kinase 1; H/R: Hypoxia/reoxygenation.