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. 2020 Apr 21;26(15):1745–1757. doi: 10.3748/wjg.v26.i15.1745

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©The Author(s) 2020. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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DNAH17-AS1 acts as a sponge for miR-432-5p in pancreatic carcinoma cells. A: Subcellular fractionation assay was performed to identify the location of DNAH17-AS1 in Hs766T cells; B: The binding sites between DNAH17-AS1 and miR-432-5p; C: Luciferase activity of miR-432-5p mimic with DNAH17-AS1-WT or DNAH17-AS1-MUT; D: MiR-432-5p expression is regulated by si-DNAH17-AS1 in Hs766T cells. E-H: Cell proliferation (E), migration (F), invasion (G), and apoptosis (H) in Hs766T cells treated with miR-432-5p mimic or si-DNAH17-AS1 and miR-432-5p inhibitor. ^aP < 0.05, ^bP < 0.01.