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. 2020 Jan 30;267(5):1420–1430. doi: 10.1007/s00415-020-09722-6

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — NfL and pNfH are increased in Friedreich’s ataxia. a Serum NfL of 30 healthy controls and 99 genetically confirmed Friedreich’s ataxia patients. b Serum pNfH in 9 healthy controls and 20 patients with Friedreich’s ataxia. **p = 0.0019, ***p = 0.0004. Data represent median and 95% confidence interval. NfL neurofilament light chain, pNfH phosphorylated neurofilament heavy chain