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. 2020 Apr 8;21(6):2580–2588. doi: 10.3892/mmr.2020.11060

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Copyright: © Xie et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Knockdown of PECAM-1 inhibits LSS-induced adhesion of monocytes to HUVECs. (A) Representative micrographs of monocyte adhesion to the HUVECs. The small round cells are adhered THP-1 monocytic cells (arrowheads). Cells of sample fields at high magnification (×200) are presented. Scale bar, 25 µm. (B) Total numbers of adherent cells in a total of three randomly selected microscopic fields. **P<0.01 vs. NC+LSS 0 min or as indicated. HUVECs, human umbilical vein endothelial cells; PECAM-1, platelet endothelial cell adhesion molecule-1; LSS, low shear stress; NC, negative control; siRNA, small interfering RNA.

Figure 3. — Knockdown of PECAM-1 inhibits LSS-induced adhesion of monocytes to HUVECs. (A) Representative micrographs of monocyte adhesion to the HUVECs. The small round cells are adhered THP-1 monocytic cells (arrowheads). Cells of sample fields at high magnification (×200) are presented. Scale bar, 25 µm. (B) Total numbers of adherent cells in a total of three randomly selected microscopic fields. **P<0.01 vs. NC+LSS 0 min or as indicated. HUVECs, human umbilical vein endothelial cells; PECAM-1, platelet endothelial cell adhesion molecule-1; LSS, low shear stress; NC, negative control; siRNA, small interfering RNA.