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. 2020 Apr 29;21:18. doi: 10.1186/s12868-020-00565-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — The molecular mechanism of COX5A over-expression on neurite outgrowth after OGD injury. a The relationship between Gstp1, Sod2, Rho-GDIa, TPI and COX5A predicted by GeneMANIA. b The mRNA expression of Rho GDP-dissociation inhibitor1. c The mRNA expression of Gstp1. d The mRNA expression of TPI. e The mRNA expression of Sod2. f The protein expression of TPI in each group. OGD oxygen glucose deprivation, NC negative control, COX5A cytochrome c oxidase subunit 5a over-expression, Gstp1 glutathione S-transferasp1, Sod2 Superoxide dismutase 2, Rho-GDIα guanine dissociation inhibitor α, TPI triosephosephate isomerase. N = 6/group. Data are presented as the mean ± SD. *p < 0.05, **p < 0.001