Table 6. Results of logistic regression analysis with the endpoint of metabolic syndrome in all participants (n = 7,750).
| Variable | Based on abdominal obesity |
Based on visceral fat obesity |
||
|---|---|---|---|---|
| Odds ratio (95% CI) | P value | Odds ratio (95% CI) | P value | |
| Model 1 | ||||
| CAVI, per 1.0 | 1.339 (1.197–1.499) | < 0.0001 | 1.188 (1.003–1.406) | < 0.05 |
| Carotid IMT, per 0.1 mm | 1.309 (1.217–1.409) | < 0.0001 | 1.439 (1.292–1.602) | < 0.0001 |
| Model 2 | ||||
| CAVI, per 1.0 | 1.338 (1.196–1.497) | < 0.0001 | 1.191 (1.006–1.409) | < 0.05 |
| Carotid IMT, per 0.1 mm | 1.311 (1.218–1.411) | < 0.0001 | 1.485 (1.288–1.598) | < 0.0001 |
CAVI, cardio-ankle vascular index; IMT, intima-media thickness; CI, confidence interval.
Endpoint of analysis fulfilled the criteria of metabolic syndrome, which is a combination of either abdominal obesity (waist circumference ≥ 85 cm for men and ≥ 90 cm for women) or visceral fat obesity (visceral fat area ≥ 100 cm2) and two other risk factors from lipid metabolism disorder (triglyceride ≥ 150 mg/dL and/or high-density lipoprotein cholesterol < 40 mg/dL), high blood pressure (systolic blood pressure ≥ 130 mmHg and/or diastolic blood pressure ≥ 85 mmHg), or high fasting blood glucose (fasting blood glucose ≥ 110 mg/dL).
Model 1 was adjusted for age, gender, and smoking status.
Model 2 was further adjusted for creatinine, plus those for Model 1.