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. 2020 Mar 27;25(3):455–466. doi: 10.1007/s12192-020-01086-0

Fig. 10.

Fig. 10

Overexpression of HSPA12B attenuated the LA-induced inhibition of wound closure. HUVECs were infected with HSPA12B adenovirus (Ad-HSPA12B) or empty adenovirus (Ad-GFP). Twenty-four hours later, cells were scratched in the presence or absence of LA (500 μM). The wound closure was measured at 48 h after scratch. **P < 0.01 vs. untreated control cells, n = 3/group. LA, α-lipoic acid