Table 2. Data collection for infarcts in tuberculous meningitis.

Subject	Data collected
Brain imaging modality	•    Non-contrast CT •    Contrast-enhanced CT •    MRI without contrast •    MRI with contrast
Timing of scan	•    Admission/baseline •    Clinical indication •    Follow–up: two to three months after initiation of treatment
Type of infarct *	•    Ischaemic •    Haemorrhagic •    Ischaemic with haemorrhagic transformation
Changes since prior brain imaging *	•    New infarct identified? •    Evolution of prior infarct?
Number of infarcts	•    Solitary •    Multifocal
In case of multifocality *	•    Bilateral •    In more than one vascular territory •    In anterior and posterior circulation territories
Vascular territories *	•    Middle cerebral artery •    Anterior cerebral artery •    Posterior cerebral artery •    Cerebellar arteries •    Vertebrobasilar perforators •    Lenticulostriate perforators •    Borderzone territory between two vascular territories     (watershed infarcts)
Location *	•    Basal ganglia •    Thalamus •    Internal capsule •    Brainstem •    Cerebellum •    Subcortical white matter •    Cerebral cortex
Size of infarct *	•    Lacunar infarcts ** •    Infarct involving >1/3 of the middle cerebral or other large artery territory •    Small punctate infarcts •    Combination of lacunar infarcts with larger areas of infarction
Evaluation of blood vessels	•    Modality used: CT angiography, MR angiography or conventional angiography •    Location of each vessel occlusion, narrowing or absence.

* More than one response allowed.

** Small subcortical infarct up to 20mm in diameter found in territories of deep penetrating arteries including basal ganglia, internal capsule, thalamus, brainstem, and corona radiata, thought to result from occlusion of a single perforating artery.

CT, computed tomography; MRI, magnetic resonance imaging.