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. 2020 May 1;18:83. doi: 10.1186/s12957-020-01859-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — NOB1 could partially reverse the roles of miR-363 on cell migration and invasion. a NOB1 re-expressed vector pcDNA-NOB1 and the miR-363 mimic were co-transfected or only transfected with miR-363 mimic in MG63 cells. b Re-expression of NOB1 could recover the migratory and invasive abilities in miR-363 overexpressing MG63 cells. *P < 0.05; **P < 0.01; ***P < 0.001