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. 2020 May 1;20:372. doi: 10.1186/s12885-020-06898-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Prdx1 significantly regulated the expression of NEDD9-Aurora A-HDAC6 in EC9706 cells. a Western-blot was used to detect the expression levels of Prdx1, NEDD9, p-Aurora A, Aurora A, HDAC6, and GAPDH in cells of each group. b Analysis of western blot signals using Image J. GAPDH was used as a loading control. c Changes of the expression of Prdx1, NEDD9, p-Aurora A, Aurora A, HDAC6, GAPDH in cells of each group. d Analysis of western blot signals using Image J. GAPDH was used as a loading control. The relative protein levels are indicated as bars. Full-length blots/gels are presented in Supplementary Fig. X. All experiments were performed in triplicate and the results are expressed as mean ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001