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. 2020 May 1;20:372. doi: 10.1186/s12885-020-06898-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Inhibition of Prdx1 decreased tumorigenesis, reduced tumor volume and weight, and promoted cilia regeneration. a Live imaging was used to detect the tumorigenesis, and the radiant efficiency and distribution of luciferase luminescence were used as indicators of tumor formation ability and tumor invasive capacity, respectively. b The radiant efficiency of luciferase luminescence in each group. c Changes of tumor weight after inhibition of Prdx1. d-e Changes of tumor volume in each group. f Immunofluorescence assay to detect the cilium marker protein (red: acetyl-α-tubulin, blue: DAPI, white arrows: cilia) in tumor tissue of each group. g Changes in the number of cells with cilia (%) in tumor tissue of each group. *P < 0.05, **P < 0.01, ***P < 0.001