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. 2010 Feb 1;201(3):346–353. doi: 10.1086/649785

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© 2010 by the Infectious Diseases Society of America

This article is made available via the PMC Open Access Subset for unrestricted re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the COVID-19 pandemic or until permissions are revoked in writing. Upon expiration of these permissions, PMC is granted a perpetual license to make this article available via PMC and Europe PMC, consistent with existing copyright protections.

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Induction of cytokines in lung epithelial cells by soluble mediators secreted by mock-infected macrophages and macrophages infected with H1N1, swine-origin influenza A/H1N1 virus California/04/2009 (S-OIV-CA), S-OIV Hong Kong/415742 (S-OIV-HK), or H5N1 (multiplicity of infection, 2). Expression of interleukin (IL)-6 (A), interferon (IFN)-α (B), macrophage inflammatory protein (MIP)-1α (C), MIP-1b (D), IFN-γ-induced protein 10 (E), monocyte chemotactic protein 1 (F), IL-1 receptor antagonist (G), IL-12p40/p70 (H), IL-8 (I), and monokine induced by IFN-γ (J) were assayed by Luminex assay. Levels of IL-2R and regulated on activation, normal T cell expressed and secreted (RANTES) were not detectable and are not shown. Means of triplicate assays are shown. C, celecoxib; CM, celecoxib plus mesalazine; M, mesalazine.