Skip to main content
. Author manuscript; available in PMC: 2020 May 7.
Published in final edited form as: Neurobiol Dis. 2020 Jan 13;137:104746. doi: 10.1016/j.nbd.2020.104746

Fig. 6.

Fig. 6.

No change in the function of corticostriatal synapses in St-GluD1 KO. A. No change was observed in the paired-pulse ratio in St-GluD1 KO; p > .999 for all intervals, Two-way ANOVA with Bonferroni’s post-hoc test. (N = 10 from 4 mice per genotype). B. No change in AMPA/NMDA ratio at corticostriatal synapses; WT 2.23 ± 0.19 vs. St-GluD1 KO 2.33 ± 0.35, p = .807, Unpaired t-test (N = 9 from 3 mice per genotype). C. No change in the evoked spike threshold; WT 3.5 ± 0.68 mA vs. St-GluD1 KO 3.13 ± 0.39 mA, p = .629, Unpaired t-test (N = 7–8 from 3 mice per genotype). All data are presented as mean ± SEM.