Table 2.
Connectivity characteristics between CN and AD participants
Measure | Group | Mean | Variance | Skewness | Kurtosis |
---|---|---|---|---|---|
Global efficiency | CN | 0.5009 | 0.0022 | −0.698 | 3.0281 |
AD | 0.5125 | 0.0017 | −0.9601 | 4.0758 | |
Local efficiency | CN | 0.7139 | 0.0033 | −0.3135 | 3.1841 |
AD | 0.7 | 0.0023 | −0.2049 | 2.9144 | |
Clustering coefficient | CN | 0.5214 | 0.0038 | 0.7414 | 3.1858 |
AD | 0.4937 | 0.0025 | 0.4945 | 2.7032 | |
Average path length | CN | 2.2716 | 0.0303 | 0.8843 | 3.2139 |
AD | 2.2031 | 0.0195 | 0.7247 | 3.0054 |
Bold indicates significance (p < 0.05). There was a significant difference in all graph theory measures between AD and CN. There was no difference between APOE4 carriers and noncarriers. We averaged metric ROI values across the brain for each subject to obtain a mean value per diagnosis. Additionally, we used variance, skewness and kurtosis to better characterize the difference in distributions between AD and CN for our respective graph theory metrics.
Key: AD, Alzheimer’s disease; APOE4, apolipoprotein E-ε4; CN, cognitively normal.