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. 2019 Sep 30;27(4):1398–1414. doi: 10.1038/s41418-019-0426-2

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© The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — PCD in the mouse heart during early embryonic development. a–d Sagittal sections of a transgenic embryo at E9.5 and of an E11.5 heart. b, d Magnification of the boxed areas: bright YFP⁺ signals (white arrows) mark PCD cells or PCD bodies. cCasp3 cleaved caspase 3, Hoechst nuclear signal, V ventricle, OFT outflow tract, RA right atrium, LA left atrium, Tr trabeculae, and iVS intraventricular septum. Bars are 200 µm (a), 10 µm (b), 100 µm (c), and 10 µm (d)