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. 2020 May 7;11(5):329. doi: 10.1038/s41419-020-2557-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Ectopic expression of miR-185-5p diminished the effects of SNORD3A overexpression on the chemosensitization of MDA-MB-231 cells to 5-FU by MTS assay at 72 h post-treatment. b, c Colony numbers of MDA-MB-231 cells with simultaneous expression interference of SNORD3A and miR-185-5p were detected by colony-formation assay (b) and soft agar colony-formation assay (c). Scale bar, 100 μm. d SNORD3A overexpression increased the chemosensitivity of MDA-MB-231-derived tumors to 5-FU, but co-expressed miR-185-5p abrogated the effect of SNORD3A. n = 5/group. Student’s t test, mean ± s.d. *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001.