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. 2020 May 7;11(5):329. doi: 10.1038/s41419-020-2557-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a SNORD3A transcript and miR-185-5p levels in breast cancer tissue samples were detected by in situ hybridization (ISH). Meis1 and UMPS protein levels in breast cancer tissue samples were detected by immunohistochemical staining (IHC) (n = 72). Scale bar, 50 μm. b Correlations between SNORD3A transcript level and Meis1 protein level, between UMPS protein level and SNORD3A transcript level, and between UMPS protein level and miR-185-5p level in breast cancer tissues were analyzed. c Kaplan–Meier analysis indicated a correlation between low SNORD3A transcript level, Meis1 protein level, or UMPS protein level and poor overall survival in breast cancer patients and a correlation between high miR-185-5p level and poor overall survival in breast cancer patients. d UALCAN (http://ualcan.path.uab.edu) showed that Meis1 expression was downregulated in breast cancer tissues. e, f Kaplan–Meier plots according to Meis1 (e) and miR-185-5p (f) expression were generated for breast cancer patient cohorts in the TCGA database. Log-rank p values are shown.