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. 2020 May 7;10:7734. doi: 10.1038/s41598-020-64672-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Mevalonate pathway inhibitors reversed fractionated-radiation–induced suppression of KLF2 and its downstream target molecules. Representative Western blot analysis and quantification of KLF2, TM, and eNOS 4 h after exposure to five fractions of 2 Gy (a) in presence or absence of atorvastatin (1 μM) or GT3 (5 μM) and (b) in presence or absence of GGTi (10 μM) (n = 3). β-actin served as a loading control. (n, number of independent experiments performed; a, significant statistical difference between nonirradiated and irradiated groups; b, significant statistical difference between fractionated irradiation and single exposure; *, p < 0.05; **, p < 0.01; ***, p < 0.001).