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. 2019 Jul 1;27(2):646–661. doi: 10.1038/s41418-019-0378-6

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© The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Loss of PDA in emc3, emc5, and emc6 mutants. a ERG recordings in wild-type flies (ey-flp rh1-GFP;FRT40A/GMR-hid CL FRT40A) showed that PDA was induced by blue light and terminated by orange light (arrows). b PDA was eliminated in ninaE^P332 flies and in c emc3^G7, e emc3^G10, g emc5^G2, and i emc6^N10 flies. PDA was restored in (d) emc3^G7;ninaE-emc3, f emc3^G10;ninaE-emc3, h emc5^G2;ninaE-emc5, and j ninaE-emc6;emc6^N10 flies. Flies ~1day after eclosion were dark-adapted for 2 min and subsequently exposed to 5-s pulses of orange (O) or blue (B) light. At least 10 flies of each genotype were tested