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. 2019 Apr 5;36(8):1252–1263. doi: 10.1089/neu.2018.5990

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Copyright 2019, Mary Ann Liebert, Inc., publishers

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FIG. 1. — A single closed head injury (CHI) results in delayed, transient mitochondrial dysfunction in the hippocampus. No significant difference was observed in oxygen consumption rate (OCR) for hippocampal mitochondria at 6, 24, or 96 h after a single CHI compared with sham injury. At 48 h post-CHI, mitochondria isolated from bilateral hippocampi demonstrated significantly lowered State III (adenosine diphosphate-mediated) respiration in the CHI group compared with sham. Other respiration states were not significantly different. State III OCR: 688.1 ± 62.9 (sham) vs. 544.7 ± 56.9 (CHI) at 48 h post-CHI. N = 6/group *p < 0.05. Bars + error bars correspond to mean + standard error of the mean.