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. 2020 Apr 16;2(2):e190114. doi: 10.1148/ryct.2020190114

Figure 6:

Figure 6:

Row 1: Motion-corrected arterial input function (AIF) (column 1) and myocardial perfusion (MP) (columns 2–4) images in a patient with a subcutaneous implantable cardioverter-defibrillator (S-ICD) show atypical perfusion defects and late gadolinium enhancement (LGE) associated with cardiac sarcoidosis. Row 2: AIF curve (column 1) and resting myocardial blood flow (MBF) maps in three short-axis planes (columns 2–4). Row 3: Wideband LGE images (columns 2–4) corroborating perfusion defects in regions of LGE. See also Movie 5 (supplement). Note the perfusion defects (green arrows) on perfusion images and MBF maps, corresponding hyperenhancement (blue arrows) on wideband LGE images, and signal voids (magenta arrows) induced by S-ICD. Regions associated with signal voids need to be interpreted with caution. Wideband saturation pulse had a full width at half maximum (FWHM) of 9.2 kHz. Wideband inversion pulse had an FWHM of 4 kHz. a.u. = arbitrary units, Gd = gadolinium.