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. 2020 May 11;22:32. doi: 10.1186/s12968-020-00609-z

Table 3.

CMR-Based Change in Diagnosis

Post-CMR diagnosis All patients (n = 208) All patients Change in diagnosis Primary prevention (n = 115) Primary prevention Change in diagnosis Secondary prevention (n = 93) Secondary prevention Change in diagnosis
Acute MI 2 2 (1) 1 1 (1) 1 1 (1)
Cardiac storage disease 1 0 (0) 1 0 (0) 0 0 (0)
ARVC 5 3 (1) 1 0 (0) 4 3 (3)
Idiopathic DCM 43 13 (6) 33 9 (8) 10 4 (4)
HCM 7 2 (1) 3 1 (1) 4 1 (1)
HHD 25 22 (11) 11 11 (10) 14 11 (12)
ICM 63 8 (4) 40 2 (2) 23 6 (6)
NCCM 1 1 (0.5) 0 0 (0) 1 1 (1)
Normal (prior LGE (+) VT ablation lesions) 4 4 (2) 1 1 (1) 3 3 (3)
Normal 21 10 (5) 5 4 (3) 16 6 (6)
Post–acute myocarditis 20 13 (6) 9 5 (4) 11 8 (9)
PPCM 2 0 (0) 2 0 (0) 0 0 (0)
Cardiac sarcoidosis 7 4 (2) 4 2 (2) 3 2 (2)
Systemic sclerosis 1 0 (0) 0 0 (0) 1 0 (0)
TOF 1 0 (0) 0 0 (0) 1 0 (0)
VHD 5 1 (0.5) 4 1 (1) 1 0 (0)
Total 208 83 (40) 115 37 (32) 93 46 (49)*

Values are n (%) for all, primary and secondary prevention groups. MI myocardial infarction; ARVC arrhythmogenic right-ventricular cardiomyopathy; DCM dilative cardiomyopathy; HCM hypertrophic cardiomyopathy; HHD hypertensive heart disease; ICM ischemic cardiomyopathy; NCCM non-compaction cardiomyopathy; LGE late gadolinium enhancement; VT ventricular tachycardia; PPCM peripartum cardiomyopathy; TOF tetralogy of Fallot; VHD valvular heart disease. * p = 0.011 for the comparison of primary versus secondary prevention