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. 2020 May 11;11(5):345. doi: 10.1038/s41419-020-2501-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — a, b VEGFA was knockdown (a) or over-expressed (b) in Caov3 and Skov3, followed by Western blotting analysis of the OC2/VEGFA/EGFL6 axis. c Schematic representation of miR-6086 molecular mechanism. miR-6086 negatively regulated tumor proliferation, metastasis and angiogenesis in ovarian cancer by down-regulating the OC2/VEGFA/EGFL6 axis in a direct or indirect manner.