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. 2020 May 11;11(5):348. doi: 10.1038/s41419-020-2525-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — PC9 cells stably expressed FTSJ1 or NC were used for in vivo study. a Growth curves of tumors in nude mice injected with stably expressed FTSJ1 of PC9 cells or control cells. b Images of xenografts derived from PC9 cells infected with lenti-FTSJ1 (upper panel) or lenti-NC (lower panel). c Tumor weight of xenografts derived from PC9 cells infected with lenti-FTSJ1 or controls. d Tumors developed from FTSJ1-overexpression cells revealed lower Ki-76 protein and higher TUNEL staining levels than that of tumors developed by lenti-NC cells, Ki-67 immunostaining; right, TUNEL staining. e qRT-PCR analysis showed that the expression level of FTSJ1 in lenti-FTSJ1-treated tumors was higher than that in lenti-NC-treated xenografts. All experiments were performed in biological triplicates with three technical replicates. Data are presented as mean ± SD. *P < 0.05; **P < 0.01; ***P < 0.001.