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. 2020 May 12;15:170. doi: 10.1186/s13018-020-01692-9

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Flii regulates tenocyte cellular migration. An in vitro scratch wound model was used to investigate the effect of differential Flii on tenocyte migration. a–c Representative images of scratch wounds in Flii^+/−, WT, and Flii^Tg/Tg tenocytes at 24 h post-wounding. d–f Representative images of scratch wounds in Flii^+/−, WT, and Flii^Tg/Tg fibroblasts at 24 h post-wounding. Magnification × 4. Scale bar = 300 μM. g Graph showing scratch closure in tenocytes expressed as percentage of original wound size. Flii^Tg/Tg tenocytes show significantly improved wound closure. h Graph showing scratch closure in fibroblasts expressed as percentage of original wound size. Flii^+/− fibroblasts show significantly improved wound closure. *p ≤ 0.05 between Flii^+/− and Flii^Tg/Tg. ^{^}p ≤ 0.05 between Flii^+/− and WT. ⁺p ≤ 0.05 between Flii^Tg/Tg and WT. Data represented as mean ± SEM. n = 6