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. 2020 May 12;18:195. doi: 10.1186/s12967-020-02362-4

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Change in circulating PCSK9 and LDL-C concentrations in human and in HFD rats. Comparisons of PCSK9 (a) and LDL-C (b) levels between tea drinker and non-tea drinkers by violin plot analysis. The vertical position of each histogram represents the relative levels of PCSK9 or LDL-C. SD rats fed with HFD for 4 weeks and administered with EGCG by i.g. at different doses for another 4 weeks. c The levels of plasma PCSK9 were analyzed by ELISA kit. d LDL-C lipid levels measured by biochemical kit. **p < 0.01, vs control, ##p < 0.01, versus HFD group (N = 6)