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. 2020 Mar 20;122(10):1467–1476. doi: 10.1038/s41416-020-0799-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Based on the analyses of genetic and clinical correlations, we consider that there is good evidence for the existence of LPD3, LPD4 and LPD5 as separate cancer categories, moderate evidence of the existence of LPD6 and LPD8 (based on alteration of expression only) and weak evidence for LPD1. The methylation column lists all genes that exhibit differential expression, and that also contain at least one locus that is differentially methylated.