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. 2020 May 13;20:165. doi: 10.1186/s12935-020-01231-w

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — miR-146a inhibitor offset function of MALAT1 in properties of HCC cell lines. HCC cells underwent simultaneous transfection of siMALAT1/siNC and/or miR-146a inhibitor/NC inhibitor for 48 h. a, b Q-PCR analysis measured the levels of MALAT1 and miR-146a expression in each group. c, d MTT assay showed the proliferation rate of HCC cell lines during 24–72 h after different transfections. e, f FC analysis showed the cell number of apoptosis in HepG2 and Huh-7 cells when treated in different ways. Data were shown as mean ± SD. *P < 0.05, **P < 0.01, ***P < 0.001, in comparison with the siNC + NC inhibitor group; ^#P < 0.05, ^##P < 0.01, ^###P < 0.001, in comparison with siMALAT1 + NC inhibitor group