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. 2020 May 13;51:67. doi: 10.1186/s13567-020-00792-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Invasion and multiplication ofS. Enteritidis in IL4I1-deficient macrophages. A Using gentamicin protection assay, with a single exception (clone 85 at 24 h compared with IL4I1^+/+ macrophages), IL4I1^+/+ and IL4I1^−/− macrophages did not show any difference in killing activity of S. Enteritidis. B Association of GFP expressing S. Enteritidis with HD11 macrophages 4 h after the infection. More IL4I1^−/− macrophages were associated with S. Enteritidis in comparison to IL4I1^+/+ macrophages. Asterisks—significantly different from the wild type IL4I1^+/+ macrophages. Data both in A and B originate from a single assay.