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. 2020 May 5;21(2):1470320320919607. doi: 10.1177/1470320320919607

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 4. — Representative immunohistochemistry staining images of kidney from DN patients (200x).

(a)–(f) AT1R, (g)–(l) AT2R, (m)–(r) MAS receptor. a,d, g, j, m, p normal controls; b, e, h, k, n, q specimens without RAS inhibitors; c, f, i, l, o, r ARB group.

ARB, AT1R blocker; AT1R, angiotensin II type 1 receptor; AT2R, angiotensin II type 2 receptor; DN, diabetic nephropathy; MASR, MAS receptor; RAS, renin–angiotensin system.